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6 Discussion Before exploring the further implications of DPP-4is in DRE, several main limitations of the current studies and this hypothesis must be acknowledged:(1)Lack of direct evidence in epilepsy: the majority of the existing evidence supporting the hypothetical “DPP-4is-gut microbiota-SCFAs-GLP-1” axis comes from cross-sectional studies (e.g., metabolic disorders, colitis, and neurodegenerative diseases), and direct causal evidence in epilepsy models is limited( Table 2 )

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